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These temporary alterations of hemodynamic and arterial stiffness are likely triggered by nicotine as an acute vasoconstrictor and also by local and circulating catecholamines which are released during nicotine consumption

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CS-induced EVs as biomarkers for the diagnosis and treatment of diseases The exposure of CS could increase the production of EVs from diverse types of cells, including endothelial cells, epithelial cells and fibroblasts, which might rely on the redox modifications of protein thiols [45, 46]

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